
Semax is a heptapeptide built from the ACTH(4–7) fragment plus a Pro-Gly-Pro tail that resists enzymatic degradation. It lacks the hormonal activity of ACTH and does not stimulate corticosteroid release. Research has examined its association with BDNF upregulation; the supporting literature is largely Russian-language, small-sample and not replicated to Western standards.
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For laboratory research use only. Not for human consumption.